oncogene
Oncogene: Gene that can cause cancer.
致癌基因: 能够导致恶性肿瘤的基因。
Oncogene: Gene that can cause cancer.
致癌基因:能够导致恶性肿瘤的基因。
Oncogene Mdm2 takes part in hepatocarcinogenesis.
罗育林，程瑞雪（导师），等.
A normal gene that has the potential to become an oncogene.
原致肿瘤基因一种有可能成为肿瘤基因的正常基因
A normal genethat has the potential to become an oncogene.
原致肿瘤基因一种有可能成为肿瘤基因的正常基因。
Expression of P53 oncogene was detected by in situ hybridization .
P53基因异常表达采用原位杂交方法。
Androgen Receptor in liver cancer progression. Oncogene or tumor suppressor?
男性激素受体与肝癌:致癌或是抑癌基因？
MEM1 is an oncogene, and its upregulation influences metastatic potential.
MEM1是致癌基因，它的上调影响癌细胞转移能力。
The stem cell leukemia( SCL) gene is a new oncogene related with leukemogenesis.
干细胞白血病(CL)因是新发现的与白血病发生有关的癌基因。
STUDY ON PATHOLOGY OF GASTRIC MUCOSA AND EXPRESSION OF ONCOGENE IN CAG MODEL WHICH COMBINED WITH SYNDROME AND DISEASE?
CAG证病结合模型的胃粘膜病理和癌基因表达研究?
Purpose To study the roles played by MDM 2 oncogene and p53 antioncogene in development of human sarcomas.
目的：研究MDM2癌基因和p53抑癌基因在人肉瘤发生中的作用。
The proto- oncogene c-fos mRNA expression was assessed using reverse transcription polymerase chain reaction(RT-PCR).
用逆转录聚合酶链式反应(RT-PCR)检测心肌细胞原癌基因c-fos mRNA的表达。
Objective: To confirm the cleavage activity of ribozyme Rz199 to oncogene K ras messenger RNA.
目的：确定人工设计的核酶Rz199对原癌基因K－rasmRNA的体外切割活性，为K－ras特异性核酶的体内应用提供依据。
Expression of N-ras Oncogene Products p21 Protein in Skin and Soft Tissue Neoplasmas.
N-ras基因产物p21蛋白在皮肤、软组织肿瘤中的表达
The data indicate that rhIL 13 enhances the expression of proto oncogene c mpl in Dami Cells.
rhIL 13增强了Dami细胞原癌基因c mpl表达 ;
Mutational activation of the BRAF oncogene is the most common genetic alteration in cutaneous melanoma.
BRAF癌基因突变的激活是皮肤黑色素瘤最常见的基因改变。
Anything from cosmic rays to radiation to diet may activate a dormant oncogene, but how remains unknown.
从宇宙射线、射到日常饮食，任何东西都可能激活一个处于静止状态下的致癌基因，但如何激活尚不为人知。
This is an example of c-myc positivity in a carcinoma.This oncogene acts via DNA transcriptional activation.
此癌基因通过使正常原癌基因数百次的复制产生一种能驱使细胞非调节性生长的蛋白。
Novel Oncogene with Kinase-domain(NOK) is a novel tumor-related gene,coding receptor like protein with a kinase domain.
是一个新的肿瘤相关基因,其结构上与受体蛋白相似,具有一个蛋白激酶结构域。
Bladder cancer occurs in coordination with oncogene active and antioncogene defective.
膀胱肿瘤的发生与癌基因的激活、抑癌基因的失活的协同作用有关。
At same time,the chan ges of the expression o f P53 oncogene and telomerase was were observed.
与此同时，P53表达和端粒酶活性发生变化。
Thompson CB,Challoner PB,Neiman PE,et al.Levels of c-myc oncogene mRNA are in variant throughout the cell cycle.Nature,1s985,314:363.
赵书芳，金岩，赵皿，等.程序性细胞死亡在牙齿发育中的作用.牙体牙髓牙周病学杂志，1999，9(1)：58.
The product of proto oncogene c Cbl has been proved as a new ubiquitin ligase (E3) of RING finger type for ubiquitin proteasome pathway.
c Cbl最近被证明是泛素 蛋白酶体 (ubiquitin proteasome)通路中的一个新的RINGFinger型泛素连接酶 (ubiquitinligase ,E3) .
Objective To investigate the inhibitory effect of RNA interference on chronic myeloid leukemia (CML) bcr/abl oncogene expression.
目的研究RNA干扰(RNAi)效应对慢性粒细胞白血病(CML)bcr/abl融合基因表达的抑制作用。
Objective: To investigate the actual role of the proto oncogene product c CBL in the signaling elicited by BCR/ABL oncoprotein.
目的 :探讨原癌基因产物c CBL在P2 10BCR/ABL激发的异常信号转导中的作用。
Objective:To approach the relationship between oncogene C-fos,K-ras and intrahep atic cholangiocarcinoma (ICC)and the significance.
目的:探讨癌基因产物C-fos、K-ras蛋白与肝内胆管癌发生的关系及其生物学意义。
Increased expression of proto oncogene c fos plays an important role in the occurence and development of fibrous dysplasia.
c?fos 癌基因的过度表达在骨纤维结构不良的发生发展中起重要作用。
The paper reviews the outcomes and developments of scientific experiments on oncogene and tumor suppression genes about human glioma.
本文试综述与胶质瘤相关的癌基因与抑癌基因的研究成果与进展。
Methods To understand the oncogene which is in relation to hepatocarcinoma and to connect the Medical Imaging with Gene diagnosis and therapy.
方法了解与肝癌有关的癌基因,将影像医学与基因诊断和治疗相结合。
Hypermethylation in the fifth oncogene, DAPK, was not associated with malignant effusion but was associated with length of time smoking.
发生超甲基化的第五个基因DAPK与恶性胸水无关，但和吸烟时间长短的有关。
Wang XJ, Zeng FF, Xu ZW, et al: Detection of tumor suppressive gene and oncogene in SO-Rb50. Human retinoblastoma cell line. Eye Science 1993; 9: 34.
冯官光,李永平,易玉珍,等:视网膜母细胞瘤细胞系裸小鼠异体移植瘤实验研究.中华眼科杂志1994;30:47.
The occurrence mechanisms on cancer including mutation of oncogene and antioncogene, effect of environmental factors were reviewed.
根据近年的研究进展,从癌基因与抑癌基因的突变及环境因素的影响等方面综述了癌症的发生机理。
The GENETIC TRANSLATION products of the fusion between an ONCOGENE and another gene. The latter may be of viral or cellular origin.
癌基因与另一个基因融合的遗传翻译产物。后者可能是病毒性或细胞源性。
The biological characteristic of carcinomas is determined both by the stromal microenvironment and the oncogene or anti-oncogene.
肿瘤的生物学特性不仅是由癌基因和抑癌基因来调控的,还依赖于由其间质等构成的微环境。
Purpose：Find the relationship between the expressions of P21 oncogene,P16 antioncogene and the biological action of the bladder cancer.
目的：探讨癌基因P21和抑癌基因P16的表达与膀胱癌生物学行为之间的关系。
The anaplastic lymphoma kinase (ALK) is the oncogene of most anaplastic large cell lymphomas (ALCL), driving transformation through many molecular mechanisms.
ALK是大部分间变性大细胞淋巴瘤（ALCL）的癌基因，它通过很多分子机制促进变异。
The development of tumor involves in many factors, such as activating of oncogen, invalidating of cancer suppressor genes and abnormal expressing of apoptosis genes.
肿瘤的发生和发展涉及原癌基因的激活、抑癌基因的失活、凋亡相关基因的异常表达等,是涉及多基因、多因素的过程。
Objective To study the expression of heat stress proteins HSP27,HSP70 and antioncogene MTS1,oncogene k ras,and their protein products in occupational lung cancer(OLC).
目的观察HSP27、HSP70与多种瘤抑制基因（MTS1）、k?ras基因及其蛋白产物在职业性肺癌（occupationallungcancer，OLC）中的联合表达的情况。
The chimeric oncogene BCR/ABL and Ph chromosome,two hall marks of CML,were detected in CML derived MSCs,normal adult MSCs,CML derived hematopoietic cells and K562 cells.
RT-PCR检测CML特异性表达的BCR/ABL基因; 对正常人、CML患者的骨髓间质干细胞进行染色体分析。
Objective: To determine the relationship between the amplification of the oncogene HER2, mdm 2 and C myc and the malignant degree,metastasis and prognosis of gastric cancer (GC).
目的 :探讨癌基因HER2、mdm 2和C myc扩增与胃癌恶性程度、转移及预后的关系。
Objective: To investigate the deletion of p53 gene and amplification of HER-2 oncogene at chromosome 17 in epithelial ovarian carcinoma(EOC) and the clinical significance.
目的:检测卵巢上皮性癌中17号染色体上野生型p53基因缺失与HER-2基因的扩增情况并探讨其临床意义。
METHODS:Serum levels of LH,FSH,PRL,T and E 2 were determined by RIA and c erbB 2 oncogene expression by immunohistochemical staining in 67 patients with gastric carcinoma.
方法 :采用放射免疫法 ,测定 67例胃癌患者血清黄体生成素 (LH ) ,卵泡刺激素 (FSH) ,催乳素 (PRL) ,睾酮 (T )及雌二醇 (E )水平 ,同时检测癌组织中c erbB 2基因表达。
CpG island demethylation facilitates gene transcription, resulting in oncogene activation, chromosome instability, mutation hotspot, and retrotransposon replacement.
CpG岛高甲基化抑制基因转录表达，导致众多抑癌基因转录沉默(transcription silencing)，蛋白表达缺郑州大学2 003年硕士研究生毕业论文胃疮中环氧合酶一2基因5’端cpG岛去甲基化与蛋白表达的关系及临床意义(中文摘要)失。
Our results suggest that the overexpression of c erbB 1 proto oncogene might be one of the important reasons that can induce keratinocytes of psoriasis to proliferate abnormally.
提示c－erbB－1的过度表达可能是导致银屑病角朊细胞异常增殖的重要原因之一。
Although inactivation of some tumor suppressor genes such as p53 and p16INK4A has been identified, no known oncogene is commonly activated in hepatocellular carcinomas.
肝癌是亚洲及非洲最常见的恶性肿瘤，其原因可能是由于这些地区肝炎病毒的流行以及经常接触肝癌的特异性致癌物质。
The miR-17-92 cluster (miR-15a, miR-16-1, miR-155, miR-17-92 cluster, miR-142) acts as an oncogene under one set of conditions and as a tumor suppressor under another.
miR-17-92簇(miR-15a,miR-16-1,miR-155,miR-17-92cluster,miR-142)是一组可能的肿瘤相关基因。
Methods: Cell was cultivated and detected with the flow cytometry and laser confocal scanning microscope methods for the oncogene PTEN expression and location.
方法：进行细胞株的培养，采用流式细胞仪和共聚焦显微镜检测方法对细胞中PTEN的表达进行定位定量检测。
With the progress of research for oncogene, it has been realized that tumourigenesis is a complicated procedure involved multiple factor and polygene mutations.
随着肿瘤发生的分子机制研究的深入,人们已逐渐认识到肿瘤的发生是一个多因素、多基因参与的复杂过程。
The mechanism of tumor dormancy is related to hindrance of angiogenesis, oncogene and antioncogene, immunocompetence, proliferation cycle, signal conduction and so on.
其发生机制可能与肿瘤血管生成受阻、癌基因与抑癌基因、宿主免疫状态、肿瘤细胞增殖周期及信号传导有关。
Thus we used MD tumor as research model and investigated the mechanism of MDV oncoginisis by researching telomere ,telomerase , tumor suppressors of chicken and MDV oncogene .
我们以MD肿瘤为模型，从端粒及端粒酶、肿瘤抑制因子、MDV的相关致瘤基因等的研究入手，对MDV的致瘤机理进行了研究。