vasospasm
That calcium antagonists on cerebral vasospasm effective.
认为钙离子拮抗剂对脑血管痉挛确实有效。
Delayed cerebral vasospasm(DCV)is one of the common complications of subarachnoid hemorrhage(SAH).
迟发性脑血管痉挛是蛛网膜下隙出血的一种常见并发症。
Furthermore, it is the first to correlate the time course of delayed cerebral vasospasm with meningitis.
此外，也第一次证实了迟发性脑血管痉挛的时程与脑膜炎之间的关系。
Objective To establish a reliable models of delayed cerebral vasospasm(DCV) after subarachnoid hemorrhage(SAH) in rats.
目的建立可靠的大鼠蛛网膜下腔出血后迟发性脑血管痉挛模型。
SGB can prevent and improve cerebral vasospasm by increasing vasorelaxant substances in plasma.
SGB可使血浆中舒血管效应的物质增多,可预防和改善脑血管痉挛。
During the procedure, 1 patients shwoed vasospasm hut no other complications such as thrombosis or angiorrhexis.
术中有1例发生血管痉挛。
The investigators infused milrinone intra-arterially into the cerebral territories compromised by the vasospasm.
研究者们将米力农注入受血管痉挛所影响的脑区的动脉中。
Objective To establish a kind of convenient,stable model of cerebral vasospasm following subarach-noid hemorrhage.
目的建立一种简便、稳定的脑血管痉挛模型。
Calcium antagonists canlunwen114 prevent vascular smooth muscle contraction caused by the vasospasm.
钙离子拮抗剂可以防止血管平滑肌收缩所导致的血管痉挛。
The basic disorder might be vascular dysfunction leading to local vasospasm and to systemic hypotension.
其基本的改变可能是血管的功能障碍导致局部的血管痉挛和低血压。
Objective: Subarachnoid hemorrhage (SAH) complicated by cerebral vasospasm (CVS) prevention and treatment methods.
摘要目的：探讨蛛网膜下腔出血(SAH)并发脑血管痉挛(CVS)的防治方法。
We suspected vasospasm in the second portion of the MCA on CTA, but could not precisely evaluate the affected lesions.
我们怀疑在CTA的第二份MCA上有血管痉挛，但是不能确切地评估病变的影响。
The subarachnoid hemorrhage from a ruptured aneurysm is more of an irritant producing vasospasm than a mass lesion.
动脉瘤破裂引起蛛网膜下腔出血的原因,血管痉挛比破裂多见。
These results indicate that K+ channel activator may play an important role for ameliorating cerebral vasospasm.
而此效果是与剂量有相关的。在经由局部和动脉注射的效果并无显著差异。
Methods The clinical data of 42 patients with delayed cerebral vasospasm induced by SAH were analyzed retrospectively.
方法回顾总结42例患者的临床资料结合文献进行分析。
Five patients (23%) had angiographically-proven vasospasm recurrence within 48 hours after the procedure.
有五名病人（占总人数的23%）经动脉造影证实在该疗程结束后48小时内复发血管痉挛。
Current smoking was the most significant independent predictor of coronary vasospasm.
抽烟是最显著的冠状动脉痉挛预测因子。
Treatment of Aneurysmal Subarachnoid Hemorrhage How to Diagnose and Manage Cerebral Vasospasm Caused by Delayed Cerebral Ischemia?
动脉瘤性蛛网膜下腔出血的治疗脑血管痉挛致迟发性脑缺血应如何诊断和处理?
The wall thickness to its inner diameter ratio was calculated on basilar artery as an indictorof cerebral vasospasm under optical microscope.
光镜下测定基底动脉的动脉壁厚度和基底动脉的内径,以其比值作为脑血管痉挛的指标。
Coronary artery vasospasm can cause a transient, abrupt, marked decrease in the diameter of an epicardial coronary artery.
摘要冠状动脉血管痉挛能造成一个暂时的，突然的，显著的心外冠状动脉内径的减少。
Sex, age, treatment modality, and use of antifibrinolytic drugs (AFD) did not influence the development of symptomatic vasospasm (all P>0.05).
入院后是否使用抗纤溶药物(AFD)治疗对症状性血管痉挛发生率无明显影响(P>0·05)。
Methods:Sixteen cases with symptomatic vasospasm after SAH were treated with isovolemic hemodilution and dobutamine and CDPC.
方法：蛛网膜下腔出血（SAH）后所致的症状性脑血管痉挛（CVS）病人16例，采用等容稀释加多巴酚丁胺和胞二磷胆碱进行治疗。
Objective To study the effects of nimodipine on symptomatic cerebral vasospasm after subarachnoid hemorrhage(SAH)in rabbits.
目的探讨尼莫地平对兔蛛网膜下腔出血(SAH)后症状性脑血管痉挛的影响。
Conclusion Internal auditory artery vasospasm can cause hearing loss.Topically use papaverine will have protection on hearing to cerfain degree.
结论内听动脉痉挛可使听觉功能明显下降，局部使用罂粟碱可预防血管痉挛，对动物听力具有保护作用。
Conclusion: Intracoronary ergonovine provocation testing was not a risky intervention procedure for the diagnosis of coronary vasospasm.
结论对诊断冠状动脉痉挛而言，冠状动脉内给予麦角新素不是危险的侵入性检查。
Objective: To observe buflomedil bydrochloride in the prevention and treatment of vasospasm after subarachnoid hemorrhage.
目的:观察盐酸丁咯地尔对蛛网膜下腔出血后血管痉挛的防治。
Cerebral vasospasm is one of the most common and serious complications of sub-arachnoid hemorrhage (SAH), and its therapeutic efficacy is not ideal.
脑血管痉挛是蛛网膜下腔出血最严重和最常见的并发症之一,临床治疗效果不理想。
Some recent studies have found that PKC plays an important role in cerebral vasospasm after cerebral ischemic injury and subarachnoid hemo rrhage.
近年来的研究发现,PKC在脑缺血性损伤和蛛网膜下腔出血后脑血管痉挛中起重要作用。
Someone said that it is not TIA if has cerebral infarct and has no evidence to support cerebral vasospasm is one of the mechanisms on TIA.
有专家认为，只要出现梗死灶就不能诊断TIA以及目前没有研究证据表明血管痉挛学说是TIA的发病机制之一。
Vasculitis, as seen in Raynaud disease , or vasospasm, as seen in decompression sickness, may interfere with extraosseous circulation.
血管炎，看到的雷诺氏病，或血管痉挛，看到的减压病，可能会干扰外流通。
Objective: To observe the effects with Nimodipine on acute and chronic cerebral vasospasm(CVS) for clinical effective application of Nimodipine.
目的：通过观察尼莫地平对蛛网膜下腔出血后急性和慢性脑血管痉挛的不同作用，为临床合理使用尼莫地平提供依据。
Conclusion Using narceine and 3H therapy is effective for patients with cerebral vasospasm after ruptured intracranial aneurysm.
结论应用罂粟堿和3H疗法是治疗脑动脉瘤破裂后迟发脑血管痉挛的一种有效的方法。
Decrease of NO in serum and increase of that in brain tissue may lead to cerebral vasospasm and ischemic brain damage respectively after SAH.
血清NO减少、脑组织NO增加分别在脑血管痉挛发生及加重脑缺血损害中起重要作用。
This case report is important because it is the first to definitively associate vasospasm with meningitis using catheter angiography.
此病例报道非常重要，因为这是第一次应用导管血管造影证实了血管痉挛与脑膜炎的相关性。
Conclusion Ischemia/reperfused injury is one of the initial factors of vasospasm and embolism after finger replantation.
结论断指(趾)再植术后缺血再灌注损伤是血管痉挛及栓塞的始动因素之一。
Objective To observe the curative effect of narceine and 3H therapy for the patients with cerebral vasospasm after ruptured intracranial aneurysm.
摘要目的观察罂粟堿和3H疗法治疗脑动脉瘤破裂后迟发性脑血管痉挛病人的疗效。
Patients with coronary vasospasm were more likely to be older, men, current smokers and to have a lower body mass index.
有冠状动脉痉挛的病人有较高的比例年长者，男性，抽烟且身体质量指数较低。
Purpose To prevent pigs from delayed cerebral vasospasm (DCV) by repeated injections of recombinant streptokinase (r SK) into lateral ventricle after subarchnoid hemorrhage (SAH).
目的 观察重组链激酶 (r SK)经侧脑室给药 ,预防蛛网膜下腔出血 (SAH)后迟发性脑血管痉挛 (DCV)的作用。
Sviri GE,Feinsod M,Soustiel JF,et al.Brain natriuretic peptide and cerebral vasospasm in aubarachnoid hemorrhage.Clinical TCD correlations[J].Stroke,2000 ;31 (1):118-22.
李倩,姜树军.脑利钠肽与蛛网膜下腔出血后脑血管痉挛[J].国外医学·脑血管疾病分册,2001;9(3):125.
INTERVENTION: Vasospasm was confirmed by angiography and transcranial Doppler sonography, and symptoms responded to hypervolemia, hypertension, and hemodilution therapy.
干涉:血管痉挛通过血管造影和经颅多普勒超声证实，针对血容量过多、高血压和血液稀释等症状进行治疗。
Coronary vasospasm was defined as a reduction in luminal diameter 70% during the provocation testing which was associated with angina and/or ST-depressive or elevated changes.
冠状动脉痉挛的定义为冠状动脉内给予麦角新素后，冠状动脉直径减少超过70%，并合并有胸间或心电图ST段下降或上升的变化。
The mechanism of the cerebral vasospasm may be related to endothelin, endothelin-derived constricting factor, endothelin-derived relaxating factor and nitric oxide.
其发生机制可能与内皮素、内皮衍生收缩因子、内皮衍生舒张因子和一氧化氮等因子有关。
Cerebral vasospasm (CVS) is a common and serious complication after subarachnoid hemorrhage (SAH).It is of great important to find effective treatments to improve the prognosis.
摘要脑血管痉挛是蛛网膜下腔出血的严重并发症，探索有效的治疗方法对改善患者的预后有着重要的意义。
Cerebral vasospasm (CVS) is an important complication after subarachnoid hemorrhage (SAH), and it is a major cause of morbidity and mortality in patients suffering from SAH.
脑血管痉挛是蛛网膜下腔出血的重要并发症,也是造成患者死亡和致残最重要的原因。
Preeclampsia-eclampsia is caused by generalized vasospasm, activation of the coagulation system, and changes in autoregulatory systems related to blood pressure control.
先兆子痫-子痫是因全身的血管痉挛，凝血系统激活，血压控制的自身调节系统改变而引起。
NPY related to NT significantly.Conclusion :NPY,secreted by platelets, may play an important role in the pathogenesis of renal hypertension and renal vasospasm in HD.
结论： 在HD过程中，血小板释放的NPY可能是造成肾性高血压和肾血管痉挛的重要病理因素。
Cerebral vasospasm (CVS) is a common and serious complication after Subarachnoid Hemorrhage (SAH).It is a primary pathogenesis lead to deformity and death.
脑血管痉挛(CVS)是蛛网膜下腔出血(SAH)的重要并发症,是SAH致死及致残的主要原因。
Szabo S,Sheth RN,Novak L,et al.Cerebrovascular reserve capacity many years after vasospasm due to aneurysmal subarachnoid hemorrhage[J].Stroke,1997,28:2479.
曹秉振，横田千晶，长谷川泰弘，等.脑循环储备力与缺血性卒中患者预后关系的研究[J].临床神经病学杂志，2001,14:200.
Objective: To investigate the reason and management of vasospasm of internal carotid artery (ICA) complicated by endovascular treatment for intracranial vascular.
摘要目的：探讨在血管内介入治疗过程中出现颈内动脉痉挛的可能原因及处理措施。
The TIA patients with carotid artery stegnosis are used CEA or CAS. 5, There are controversy about concept on TIA with infarct and TIA due to cerebral vasospasm.
颅外颈动脉粥样硬化性狭窄，CEA或CAS处理，颅外椎基底动脉/颅内动脉粥样硬化性狭窄，CAS处理。5、梗死型和血管痉挛学说尚有争议。